For heat stress
Heat stress in swine triggers a severe physiological cascade that extends beyond hyperthermia. A primary adaptation is the voluntary reduction of feed intake to decrease metabolic heat production. Concurrently, blood flow is redirected from the gastrointestinal tract to the skin to facilitate heat dissipation, leading to splanchnic hypoxia. This compromises intestinal barrier integrity, allowing the translocation of endotoxins like lipopolysaccharides (LPS) into the bloodstream. The subsequent systemic inflammatory response and overproduction of reactive oxygen species (ROS) induce a state of severe oxidative stress, which damages cells and diverts energy from productive functions.
Effective mitigation requires targeted nutritional additives that address these specific physiological failures. The strategy is to reinforce the animal’s defenses and support homeostasis. Key functional additives include phytogenics, particularly polyphenols, provide broad-spectrum ROS scavenging activity and gut barrier support.
An integrated mitigation strategy
A multi-faceted nutritional approach is essential. Combining these additives creates a robust defense by simultaneously addressing gut barrier failure, oxidative stress, and cellular disruption. This integrated fortification enhances swine resilience, preserving health and performance during periods of thermal challenge.

Ruminants
Poultry
Swine
Fishes / aquaculture
Pet