AviPremium® is a complete range of products based on tributyrin, a triglyceride of butyric acid that helps to maintain the integrity of the intestinal.
Species
Ruminants
The need to guarantee ruminant absorption of a correct amount of nutrients in the intestine cannot disregard the degradation by the rumen microflora. For over 30 years our scientific research has set the goal to improve the rumen bypass so that a lower concentration of active ingredients in the diet leads to maximizing performance.
- Microtinic®Omega
- Microtinic®E
- Microtinic®AD3E
- Microtinic®PP
- Microtinic®C
- Microtinic®B12
- Microtinic®B-Complex
- AviPremium®
- SmartSel®
- Ruprocol®
- MecoVit®
- Relys®
- Timet®
AviPremium®
INNOVATIVE
AviPremium® products are based on tributyrin, a triacylglycerol of butyric acid (EU Reg. 1831/2003; FDA 21 CFR 184.1903). Tributyrin is the most concentrated derivate of butyric acid on the market. The tributyrin in AviPremium® is more stable and has superior organoleptic characteristics than butyric acid. AviPremium® products are an alternative to Na-butyrate. In this product, the butyric acid from tributyrin is chemically protected and delivered where butyric acid can be effective. The esterification with glycerol allows the butyric acid to be released in the intestine due to the action of pancreatic lipases. Its microencapsulation allows further advantages and efficacy at the intestinal level.
UNIQUE PRODUCTS
The full range of products is composed of: AviPremium® L based on liquid tributyrin (85% butyric acid). AviPremium® D, a concentrated source of tributyrin in powder form (55% butyric acid). AviPremium® S and P where the tributyrin is microencapsulated within a vegetable lipid matrix according to the digestive characteristics of swine and poultry respectively (25% butyric acid).
Latest articles from the Press Room
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Our findings in Holstein calves are early evidence that dietary microencapsulated OA/PB feeding is a means to partially restore feed intake and average daily gain post-weaning when challenged by heat exposure.
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Heat stress develops with methyl donor deficiency in parallel with an impaired N metabolism. The supplementation of OA/PB improves the remethylation capacity in the liver. On-going transcriptomic analyses will provide a better understanding of the hepatic metabolism of dairy cows exposed to heat stress.